3rd World Brain Congress

Alzheimer's disease (AD) may be a progressive neurodegenerative disease characterized memory loss and psychological feature impairment. It’s caused by conjugation failure and excessive accumulation of misfolded proteins. To date, most advanced clinical trials on specific AD-related pathways have unsuccessful largely thanks to an oversized variety of neurons lost within the brain of patients with AD. Somatic cell treatment has been thriving in AD animal models. Recent diagnosis studies on somatic cell medical care for AD have verified to be promising. Cell replacement therapies, like human embryonic stem cells or induced pluripotent stem cell–derived neural cells, have the potential to treat patients with AD, and human clinical trials square measure in progress during this regard. AD may be a progressive chronic unwellness that sometimes begins many years before identification. Therefore, a person's brain is severely broken by the time symptoms or signs seem and an oversized v...

In a myelinated nerve fiber, the conduction of impulse is faster due to the presence of Myelin sheath and node of Ranvier.   As the rate of impulse conduction in a nervous system depends on the diameter of a nerve fiber and the presence or absence of myelin sheath.  Neurons with myelin sheath conduct impulse much faster than those without the presence of myelin. Schwann cells (or oligodendrocytes) are located at various regular intervals along the axons of the neuron. Between the areas of myelin are non-myelinated areas present, called as the nodes of Ranvier. Because fat or lipid molecules (myelin) acts as an insulator, a membrane coated with myelin does not conduct an impulse.  So, in a myelinated neuron, action potentials occur only along with the nodes and, therefore, impulses jump over the areas of myelin, going from node to node in a process called as saltatory conduction. Action potential velocity Brain cells are also called neurons which ...